Study on Thrombocytic Ultrastructure and Functions of Blood Stasis Syndrome in Liver Disorders.

doi:10.13288/j.11-2166/r.1996.12.020

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Study on Thrombocytic Ultrastructure and Functions of Blood Stasis Syndrome in Liver Disorders.

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- Study on Thrombocytic Ultrastructure and Functions of Blood Stasis Syndrome in Liver Disorders.
- Journal of Traditional Chinese Medicine Issue 12, Pages: 740-741(1996)
- 作者机构：
  
  1. 湖北中医学院肝病研究所
  2. 湖北中医学院肝病研究所武汉市武昌区花园山4号,430061
- 作者简介：
- 基金信息：
- DOI：10.13288/j.11-2166/r.1996.12.020
  CLC： R256.4
- Published：1996
- 稿件说明：
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[1]张赤志,张建军.肝病血瘀证患者血小板超微结构和功能的研究[J].中医杂志,1996(12):740-741+708.

Study on Thrombocytic Ultrastructure and Functions of Blood Stasis Syndrome in Liver Disorders.[J]. Journal of Traditional Chinese Medicine1996, (12): 740-741.
[1]张赤志,张建军.肝病血瘀证患者血小板超微结构和功能的研究[J].中医杂志,1996(12):740-741+708. DOI： 10.13288/j.11-2166/r.1996.12.020.

Study on Thrombocytic Ultrastructure and Functions of Blood Stasis Syndrome in Liver Disorders.[J]. Journal of Traditional Chinese Medicine1996, (12): 740-741. DOI： 10.13288/j.11-2166/r.1996.12.020.

摘要

通过对肝病血瘀证患者血小板超微结构和聚集功能研究。结果表明:盘状细胞较少

伪足变形较多

聚集倾向的血小板增多

糖原增多的血小板比例增大;大型血小板比例增多。可见血小板粘附红细胞、白细胞及淋巴细胞现象;血小板内颗粒减少

甚至相互融合

辨认不清;管道系统扩张并增生。易见异常颗粒

大小与α-颗粒相似

有界膜包绕

内容物为胞浆及代谢产物。其在ADP诱导下

聚集体占70.28％

多数细胞间有桥状联系而模糊不清

甚至细胞间隙消失

细胞膜融。以上变化均较对照组明显。提示血小板超微结构的变化和聚集功能改变

是造成肝病血瘀证的原因之一。

Abstract

The results of this study revealed that there was less discoid cell and more pseudopodium

more thrombocytes tended to agglutinate with increased proportion of hyper - glycogenic platelets. There were also platelets sticking to RBC. WBC and lymphocytes. The glanules within the platelates were decreased or even fused together

unable to differentiate them. The vascular system were dilated and proliferated. Abnormal granules were seen with its size similar to a - granules

covered with membrane

with cytoplasm and metabolites inside. Under the revulsion of ADP

there was 70. 28% agglutining body. Most intercellular spaces revealed unclear bridge - form connection or even disappeared with fusion of cellular membrane. All these changes were more distinctive than that in the control group

demonstrating the changes in ultrasturcture and agglutination function might be one of the causes of blood stasis in hepatopathy.

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