Differential Diagnosis of Cold - heat Syndrome in Duodena] Ulcer and Helicobacter Pylori - inflammation-interleukin-8

doi:10.13288/j.11-2166/r.1998.10.025

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Differential Diagnosis of Cold - heat Syndrome in Duodena] Ulcer and Helicobacter Pylori - inflammation-interleukin-8

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- Differential Diagnosis of Cold - heat Syndrome in Duodena] Ulcer and Helicobacter Pylori - inflammation-interleukin-8
- Journal of Traditional Chinese Medicine Issue 10, Pages: 616-618(1998)
- 作者机构：
  
  1. 北京医科大学第一医院中西医结合研究所
  2. 北京医科大学第一医院消化科
  3. 北京医科大学第一医院中西医结合研究所北京西城区,100034
- 作者简介：
- 基金信息：
- DOI：10.13288/j.11-2166/r.1998.10.025
  CLC： R259.7
- Published：1998
- 稿件说明：
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[1]张学智,纪保安,陈宝雯,贾博琦,谢竹藩.幽门螺杆菌、炎症、白介素—8与十二指肠溃疡的寒热辨证[J].中医杂志,1998(10):616-618+580.

Differential Diagnosis of Cold - heat Syndrome in Duodena] Ulcer and Helicobacter Pylori - inflammation-interleukin-8[J]. Journal of Traditional Chinese Medicine1998, (10): 616-618.
[1]张学智,纪保安,陈宝雯,贾博琦,谢竹藩.幽门螺杆菌、炎症、白介素—8与十二指肠溃疡的寒热辨证[J].中医杂志,1998(10):616-618+580. DOI： 10.13288/j.11-2166/r.1998.10.025.

Differential Diagnosis of Cold - heat Syndrome in Duodena] Ulcer and Helicobacter Pylori - inflammation-interleukin-8[J]. Journal of Traditional Chinese Medicine1998, (10): 616-618. DOI： 10.13288/j.11-2166/r.1998.10.025.

摘要

选用48例十二指肠溃疡患者进行寒热辨证

根据辨证分寒证组、无明显寒热证组及热证组

并研究其与幽门螺杆菌（HP）、急慢性炎症程度、白细胞介素—8（IL—8）的相关性。结果显示:热证组HP感染及感染密度明显高于寒证组（P<0.001）;急、慢性炎症程度在热证组较寒证组重;IL—8在热证组胃粘膜中明显高于寒证组及无明显寒热证组（P<0.05）。提示溃疡病寒热证型与HP感染、急慢性炎症程度明显相关。IL—8可能是寒热证形成的机制之一。

Abstract

Forty - eight cases of duodenal ulcer were differentially diagnosed for their syndrome types as heat type

cold type and non -heat-non-cold type

with their coincidence with helicobactor - pylori （HP）

acute and chronic inflammatory degrees

IL -8. Results revealed that the incidence of HP infection and its severity were higher in the heat group than in the cold group. The IL - 8 in the stomach mucosa in the heat group was markedly higher than in the other groups （P<0. 05）

demonstrating that the cold-heat types were closely related to the infection of Hp. while IL - 8 might be one of the molecular mechanisms in the formation of cold - heat syndrome types formation.

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