Discussion on Coronary Microcirculation Disorder after Myocardial Ischemia Reperfusion Based on “Collaterals-Sweat Pore Qi and Fluid” Theory

SHI Haoyue; SHANG Juju; LIU Hongxu; QIU Shenglei; LI Sinai; XING Wenlong; FAN Yingbing; YANG Linjing

doi:10.13288/j.11-2166/r.2023.18.006

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Discussion on Coronary Microcirculation Disorder after Myocardial Ischemia Reperfusion Based on “Collaterals-Sweat Pore Qi and Fluid” Theory

更新时间：2023-09-12

- Discussion on Coronary Microcirculation Disorder after Myocardial Ischemia Reperfusion Based on “Collaterals-Sweat Pore Qi and Fluid” Theory
- Journal of Traditional Chinese Medicine Vol. 64, Issue 18, Pages: 1862-1865(2023)
- 作者机构：
  
  1.首都医科大学附属北京中医医院，北京市东城区美术馆后街23号，100010
  2.北京市中医药研究所
  3.北京市大兴区黄村镇社区卫生服务中心
  4.首都医科大学
- 作者简介：
- 基金信息：
- DOI：10.13288/j.11-2166/r.2023.18.006
  CLC：
- Received：15 March 2023，
  
  Revised：2023-06-23，
  
  Published：17 September 2023
- 稿件说明：
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石皓月,尚菊菊,刘红旭等.基于“络脉-玄府气液”探讨心肌缺血再灌注治疗后冠状动脉微循环障碍[J].中医杂志,2023,64(18):1862-1865.

SHI Haoyue,SHANG Juju,LIU Hongxu,et al.Discussion on Coronary Microcirculation Disorder after Myocardial Ischemia Reperfusion Based on “Collaterals-Sweat Pore Qi and Fluid” Theory[J].Journal of Traditional Chinese Medicine,2023,64(18):1862-1865.
石皓月,尚菊菊,刘红旭等.基于“络脉-玄府气液”探讨心肌缺血再灌注治疗后冠状动脉微循环障碍[J].中医杂志,2023,64(18):1862-1865. DOI： 10.13288/j.11-2166/r.2023.18.006.

SHI Haoyue,SHANG Juju,LIU Hongxu,et al.Discussion on Coronary Microcirculation Disorder after Myocardial Ischemia Reperfusion Based on “Collaterals-Sweat Pore Qi and Fluid” Theory[J].Journal of Traditional Chinese Medicine,2023,64(18):1862-1865. DOI： 10.13288/j.11-2166/r.2023.18.006.

摘要

心肌缺血再灌注治疗后出现的冠状动脉微循环障碍是冠心病治疗领域的突出问题。根据“络脉-玄府气液”与冠状动脉微循环生理上的共通性，结合心肌缺血再灌注损伤的病程演变过程，认为“心络瘀阻，玄府郁闭”是冠状动脉微循环障碍的基本病机，其病机演变可分为3个阶段。1）心肌缺血期：心络瘀阻，玄府空虚；2）再灌注治疗期：虚风内起，络脉拘急或微小心络痹阻；3）冠状动脉微循环障碍期：玄府郁闭，郁而化热，气液失宣。治则分别为以补助通、补虚逐瘀，风血同治、熄风解痉，清热凉血、宣畅气液，同时应注重心肺同治，心肝同调。

Abstract

Coronary microcirculation disorder after myocardial ischemia reperfusion （MIR） is a prominent problem in the treatment of coronary heart disease. According to the physiological commonality between “collaterals-sweat pore

and fluid” and coronary microcirculation， and the evolution of the course of MIR， it is believed that “heart collateral stasis obstruction， sweat pore constraint and block” is the cause of coronary microcirculation disorder. The evolution of the pathogenesis can be divided into three periods. During the myocardial ischemia period， the pathogenesis is heart collaterals obstruction and sweat pores empty， while during the ischemia reperfusion period， it is internal formulation of deficiency wind， spasms of collaterals or slight heart collaterals obstruction； in the coronary microcirculation disorder period， sweat pores constraint and block， constraint transforming into heat，

and fluid failing to diffuse are the pathogenesis. The corresponding treatment principle is assisting dredge with supplementation， and supplementing deficiency to dispel stasis； treating wind and blood simultaneously， and extinguishing wind to arrest convulsion； clearing heat and cooling blood， and diffusing

and unblocking

and fluid. Moreover， it is recommended to treat the heart and lungs simultaneously， and regulate the heart and liver at the same time.

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references

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