The results of this study revealed that there was less discoid cell and more pseudopodium

more thrombocytes tended to agglutinate with increased proportion of hyper - glycogenic platelets. There were also platelets sticking to RBC. WBC and lymphocytes. The glanules within the platelates were decreased or even fused together

unable to differentiate them. The vascular system were dilated and proliferated. Abnormal granules were seen with its size similar to a - granules

covered with membrane

with cytoplasm and metabolites inside. Under the revulsion of ADP

there was 70. 28% agglutining body. Most intercellular spaces revealed unclear bridge - form connection or even disappeared with fusion of cellular membrane. All these changes were more distinctive than that in the control group

demonstrating the changes in ultrasturcture and agglutination function might be one of the causes of blood stasis in hepatopathy.