中国中医科学院广安门医院,北京市西城区北线阁5号,100053
drlinhongsheng@163.com
收稿:2025-06-23,
修回:2025-07-24,
纸质出版:2025-11-02
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谭滢,韩宝瑾,钟毅薇等.基于湿浊三焦传变探讨肺癌胸腔积液的治疗思路[J].中医杂志,2025,66(21):2287-2291.
TAN Ying,HAN Baojin,ZHONG Yiwei,et al.Therapeutic Approach to Malignant Pleural Effusion in Lung Cancer Based on the Transmission and Transformation of Damp Turbidity through the Sanjiao (三焦)[J].Journal of Traditional Chinese Medicine,2025,66(21):2287-2291.
谭滢,韩宝瑾,钟毅薇等.基于湿浊三焦传变探讨肺癌胸腔积液的治疗思路[J].中医杂志,2025,66(21):2287-2291. DOI: 10.13288/j.11-2166/r.2025.21.017.
TAN Ying,HAN Baojin,ZHONG Yiwei,et al.Therapeutic Approach to Malignant Pleural Effusion in Lung Cancer Based on the Transmission and Transformation of Damp Turbidity through the Sanjiao (三焦)[J].Journal of Traditional Chinese Medicine,2025,66(21):2287-2291. DOI: 10.13288/j.11-2166/r.2025.21.017.
认为“湿浊蓄于下焦-逆传三焦-上凌肺络”为肺癌胸腔积液的核心病机,并据此阐释肺癌胸腔积液的形成路径:湿浊先蓄于下焦,肾与膀胱气化失司为病源;继而逆传三焦,少阳枢机不利为枢纽;最终上凌肺络,胸阳痹阻致水饮停聚。据此确立其辨治原则:温肾化气以固根本,通过温补肾阳、助膀胱气化阻断湿浊生成;疏利三焦以截传变,调畅水道阻止湿浊上犯;开泄上焦以逐饮邪,缓解肺络壅滞之症,为临床辨治恶性胸腔积液提供新思路。
This paper proposes that the core pathogenesis of malignant pleural effusion in lung cancer lies in "accumulation of damp turbidity in the lower
sanjiao
(三焦), retrograde transmission through the
sanjiao
, and upward invasion of the lung collaterals". This theory elucidates the pathophysiological process of effusion formation as follows: damp turbidity first accumulates in the lower
sanjiao
, where dysfunction of the kidney and bladder in
qi
transformation serves as the source of disease; it then transmits retrogradely through the
sanjiao
, with stagnation of the
shaoyang
pivot acting as the key link; finally, the damp turbidity ascends to the lung collaterals, where obstruction of chest
yang
leads to fluid retention in the thoracic cavity. Accordingly, the therapeutic principles are established as follows: warming the kidney and promoting
qi
transformation to strengthen the root, by tonifying kidn
ey
yang
and faci-litating bladder
qi
transformation to block the generation of damp turbidity; dredging and unblocking the
sanjiao
to interrupt its pathological transmission, by regulating water pathways and preventing upward encroachment of damp turbidity; and opening and dispersing the upper
sanjiao
to eliminate retained fluids, by relieving obstruction of the lung collaterals. This approach provides a novel perspective for the syndrome differentiation and treatment of malignant pleural effusion in lung cancer.
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